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  • Cell Research
    Advanced Online Publication Current Issue Top 10 VOLUME 22 ISSUE 11 11 2012 1511 1511 Sanofi Cell Research Outstanding Paper Award of 2011 Sanofi Cell Research Outstanding Paper Award of 2011 Deputy Editor in Chief Cell Research Shanghai Institutes for Biological Sciences Chinese Academy of Sciences Shanghai 200031 China Correspondence Dangsheng Li E mail dsli sibs ac cn Cell Research 2012 22 1511 doi 10 1038 cr 2012 153 published

    Original URL path: http://www.cell-research.com/arts.asp?id=81 (2016-02-14)
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  • Cell Research
    Boston MA 02215 USA Correspondence Jia huai Wang E mail jwang red dfci harvard edu The inside out signaling of integrins regulates the ligand binding affinity of the cell surface receptors in response to changes in the environment for cell survival The specific binding to the cytoplasmic tail of integrin s β subunit by the intracellular protein talin is the key step of inside out signaling A pull push mechanism

    Original URL path: http://www.cell-research.com/arts.asp?id=82 (2016-02-14)
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  • Cell Research
    Russell Department of Molecular Medicine Mayo Clinic Rochester Minnesota 55905 USA Correspondence Stephen J Russell E mail sjr mayo edu The rapid mutation of RNA viruses allows for the acquisition of resistance to drugs directly targeting viral proteins Therefore a novel approach to the development of antivirals centers on targeting host factors critical to viral replication A recent report has brought to light the potential for RNA viruses to also

    Original URL path: http://www.cell-research.com/arts.asp?id=83 (2016-02-14)
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  • Cell Research
    Center San Diego State University San Diego CA 92182 USA Correspondence Roberta A Gottlieb E mail robbieg sciences sdsu edu The link between impaired autophagic flux autophagus interruptus damaged mitochondria and myocardial inflammation has been further tightened with the recent paper by Oka and colleagues in which failure to degrade mitochondrial DNA exacerbated myocardial inflammation in the context of pressure overload Using mice with cardiac specific deletion of the lysosomal

    Original URL path: http://www.cell-research.com/arts.asp?id=84 (2016-02-14)
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  • Cell Research
    2 and Kenneth R Chien 1 2 1 Department of Stem Cell and Regenerative Biology Harvard University Cambridge MA 02138 USA 2 Cardiovascular Research Center Massachusetts General Hospital Boston MA 02114 USA Correspondence Kenneth R Chien E mail kchien harvard edu The term lineage reprogramming is typically used to describe the conversion of one differentiated somatic cell type into another without transit through a pluripotent intermediate Two recent reports in

    Original URL path: http://www.cell-research.com/arts.asp?id=85 (2016-02-14)
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  • Cell Research
    of Massachusetts Medical School Worcester MA 01605 USA 2 Program in Molecular Medicine University of Massachusetts Medical School Worcester MA 01605 USA Correspondence Ingolf Bach E mail ingolf bach umassmed edu One of the two X chromosomes in cells of female mammals is transcriptionally silenced in a process known as X chromosome inactivation XCI Initiation of XCI is regulated by the ubiquitin ligase Rnf12 RLIM but the mechanisms by which

    Original URL path: http://www.cell-research.com/arts.asp?id=86 (2016-02-14)
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  • Cell Research
    of Molecular Oncology Institute for Genetic Medicine Hokkaido University North 15 West 7 Kita ku Sapporo Hokkaido 060 0815 Japan Correspondence Yasuyuki Fujita Tel 81 11 706 5530 E mail yasu igm hokudai ac jp It is not clearly understood what happens at the interface between normal and transformed epithelial cells at the first step of carcinogenesis A recent study reveals that the organized epithelial structure suppresses clonal expansion of

    Original URL path: http://www.cell-research.com/arts.asp?id=87 (2016-02-14)
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  • Cell Research
    162 8655 Japan Correspondence Michihiro Matsumoto E mail mmatsumoto ri ncgm go jp During fasting dephosphorylation dependent activation of the CREB coactivator CRTC2 by glucagon is crucial for activation of the hepatic gluconeogenic program but the molecular mechanism by which hormones regulate CRTC2 activation remains unclear A recent report in Nature showed that PKA dependent phosphorylation of the inositol 1 4 5 trisphosphate receptor InsP3R induces Ca2 mobilization leading to

    Original URL path: http://www.cell-research.com/arts.asp?id=88 (2016-02-14)
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